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https://www.sciencedirect.com/science/article/pii/S0168365905003329
The transfected cells showed a significant extent of apoptosis by rev-caspase-3. This study suggests the potential of using folate-receptor-mediated delivery of rev-caspase-3 gene for inducing tumor cell death in a target-specific manner.Author: Kyung Chul Cho, Ji Hoon Jeong, Hyun Jung Chung, Cheol O Joe, Sung Wan Kim, Tae Gwan Park
https://www.researchgate.net/publication/7621237_Folate_receptor-mediated_intracellular_delivery_of_recombinant_caspase-3_for_inducing_apoptosis
Folate receptor-mediated intracellular delivery of recombinant caspase-3 for inducing apoptosis Article in Journal of Controlled Release 108(1):121-31 · December 2005 with 19 Reads
https://www.sciencedirect.com/science/article/pii/S0142961209004086
The use of folate-PEG-grafted-hybranched-PEI nonviral vector for the inhibition of glioma growth in the rat. ... S. Wan Kim, T. Gwan ParkFolate receptor-mediated intracellular delivery of recombinant caspase-3 for inducing apoptosis. J Control Release, 108 (2005), pp. 121-131.Author: Bing Liang, Ming-Liang He, Chu-yan Chan, Yang-chao Chen, Xiang-Ping Li, Yi Li, Dexian Zheng, Marie C...
https://www.deepdyve.com/lp/elsevier/the-use-of-folate-peg-grafted-hybranched-pei-nonviral-vector-for-the-kfcur01tDC
Aug 01, 2009 · Read "The use of folate-PEG-grafted-hybranched-PEI nonviral vector for the inhibition of glioma growth in the rat, Biomaterials" on DeepDyve, the largest online rental service for scholarly research with thousands of academic publications available at your fingertips.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2909748/
May 28, 2010 · Caspase-3 activation increased by 33 ± 21 % immediately (at 0 min) after a 10 min treatment with NMDA, although not reaching statistical significance. Active caspase-3 continued to rise, peaking at ~30 min after NMDA (213 ± 23% of baseline, n=4, p<0.01), before gradually returning to pre-treatment levels (Figure 6A, B).Author: Zheng Li, Jihoon Jo, Jie-Min Jia, Shih-Ching Lo, Daniel J. Whitcomb, Song Jiao, Kwangwook Cho, Morga...
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3564970/
2.2 Intrinsic Apoptosis Pathway. A variety of cellular stresses activate the transcription factor p53, which is known as the tumor suppressor gene. p53 inhibits tumorigenesis by triggering apoptosis in stressed or damaged cells and operates through several proposed mechanisms including the transactivation of pro-apoptotic factors [].One of the important functions of p53 is its role in the Bcl ...Author: Hongmei Li, Chris E. Nelson, Brian C. Evans, Craig L. Duvall
https://www.researchgate.net/publication/324091555_Inducing_cell_death_in_vitro_in_cancer_cells_by_targeted_delivery_of_Cytochrome_c_via_a_Transferrin_conjugate
Inducing cell death in vitro in cancer cells by targeted delivery of Cytochrome c via a Transferrin conjugate Article (PDF Available) in PLoS ONE 13(4) · March 2018 with 273 Reads
http://www.jbc.org/content/279/1/469.full
Jan 02, 2004 · Substantial progress over the past decade has validated the insulin-like growth factor binding proteins (IGFBPs) 1 as unique intrinsic effectors of cellular growth and apoptosis, properties that can be unrelated to their ability to bind IGFs (for review see Ref. 1).Several lines of evidence support insulin-like growth factor binding protein-3 (IGFBP-3) as a biological mediator of cancer cell ...Author: Kuk-Wha Lee
https://www.cell.com/cell/fulltext/S0092-8674(10)00372-7
May 28, 2010 · NMDA receptor-dependent synaptic modifications, such as long-term potentiation (LTP) and long-term depression (LTD), are essential for brain development and function. LTD occurs mainly by the removal of AMPA receptors from the postsynaptic membrane, but the underlying molecular mechanisms remain unclear. Here, we show that activation of caspase-3 via mitochondria is required …Author: Zheng Li, Jihoon Jo, Jie-Min Jia, Shih-Ching Lo, Daniel J. Whitcomb, Song Jiao, Kwangwook Cho, Morga...
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