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https://www.ncbi.nlm.nih.gov/pubmed/3524919
Increased delivery of NaCl to the macula densa did not alter total renin, but decreased inactive renin from 30% to 0, crystalline core-containing cells from 33% to 14% and decreased the percentage of granules with crystalline cores from 12% to 2.2%.Author: T. Wong, T. O. Morgan, D. Alcorn, G. B. Ryan
https://www.sciencedirect.com/topics/medicine-and-dentistry/macula-densa
Increased sodium chloride delivery to the macula densa causes release of adenosine, which then binds to the afferent arteriole causing vasoconstriction. 80 This vasoconstriction subsequently decreases GFR. The opposite response occurs because of decreases in sodium chloride delivery to the macula densa.
https://www.sciencedirect.com/topics/veterinary-science-and-veterinary-medicine/macula-densa
Macula-densa cells may signal granular cells to inhibit renin release by secreting adenosine and/or ATP across the basolateral membrane in response to increased sodium delivery [96, 101, 102]. The precise mechanism of this effect is not clear. Adenosine stimulates granular-cell A 1 and A 2 receptors.
https://deepblue.lib.umich.edu/bitstream/handle/2027.42/24023/0000272.pdf;sequence=1
sociated with renin synthesis and release [ 1, or by some function of sodium delivery to 8-10 1. However, the precise role in the reg- the macula densa cells-macula densa the- ulation of this hormone is unknown. Con- ory [ 111. The latter appears to have a greater
https://www.ahajournals.org/doi/pdf/10.1161/CIRCULATIONAHA.113.005081
tubule glucose and sodium reabsorption, which leads to increased sodium delivery to the macula densa. This condition restores TGF via appropriate modulation of arteriolar tone (eg, afferent vasoconstriction), which in turn reduces renal plasma flow and hyperfiltration.
https://onlinelibrary.wiley.com/doi/10.1002/ejhf.35
In CHF, because of increased fractional sodium reabsorption in the proximal tubules, chloride delivery to the macula densa is reduced, which causes a vicious cycle of worsening congestion and harmful neurohumoral activation that might ultimately drive disease progression (Figure 3B).Author: Frederik H. Verbrugge, Matthias Dupont, Paul Steels, Lars Grieten, Quirine Swennen, W.H. Wilson Tang...
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