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https://www.ahajournals.org/doi/full/10.1161/01.CIR.101.25.2981
Postinfarction remodeling has been arbitrarily divided into an early phase (within 72 hours) and a late phase (beyond 72 hours). The early phase involves expansion of the infarct zone, 5 which may result in early ventricular rupture or aneurysm formation.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2504336/
Long-term outcome after MI can be largely be defined in terms of its impact on the size and shape of the left ventricle (i.e., LV remodeling). Three major mechanisms contribute to LV remodeling: 1) early infarct expansion, 2) subsequent infarct extension into adjacent noninfarcted myocardium, and 3) late hypertrophy in the remote LV.
https://www.ahajournals.org/doi/pdf/10.1161/01.CIR.0000036014.90197.FA
and prognostic value of LV remodeling after primary PTCA are still to be clarified. Methods and Results—In 284 consecutive patients with AMI treated with primary PTCA, serial echocardiographic and angiographic studies, within 24 hours (T1), at 1 (T2) and 6 months (T3) after AMI were performed.
https://www.sciencedirect.com/topics/medicine-and-dentistry/heart-ventricle-remodeling
While coronary reperfusion within 30 minutes after onset of an STEMI can limit infarct size, LV dilative remodeling and dysfunction, few patients present that early and adverse LV remodeling occurs with STEMI reperfused ≥ 90 minutes from the onset and results in persistent LV dysfunction and HF, 31–34 which plague STEMI survivors worldwide. 31–35 The major players …
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