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https://www.sciencedirect.com/topics/medicine-and-dentistry/macula-densa
Macula-densa cells may signal granular cells to inhibit renin release by secreting adenosine and/or ATP across the basolateral membrane in response to increased sodium delivery [96, 101, 102]. The precise mechanism of this effect is not clear. Adenosine stimulates granular-cell A 1 and A 2 receptors.
https://www.ncbi.nlm.nih.gov/pubmed/3524919
Increased sodium in the diet and increased delivery of NaCl to the macula densa decreased the proportion of renin present in the inactive form and decreased the proportion of crystalline cores. These coincidental alterations suggest that crystalline cores contain inactive renin and suggest that the delivery of sodium to the macula densa activates renin.Author: T. Wong, T. O. Morgan, D. Alcorn, G. B. Ryan
https://diabetes.diabetesjournals.org/content/68/6/1109
Jun 01, 2019 · Sodium–glucose cotransporter 2 (SGLT2) inhibitors are the most recently approved class of diabetes drugs. Unlike other agents, SGLT2 inhibitors act on the kidney to promote urinary glucose excretion. SGLT2 inhibitors provide multiple benefits, including …Author: Amber L. Beitelshees, Bruce R. Leslie, Simeon I. Taylor
https://deepblue.lib.umich.edu/bitstream/handle/2027.42/24023/0000272.pdf;sequence=1
tion of sodium delivery to the macula densa cells [ 111, or both [5]. However, the macula densa theory has a greater number of pro- ponents. Macula densa cells have been reported to contain cilia and may be capable of detecting physical stimuli [6]. Physically, the macula
https://www.kidney-international.org/article/S0085-2538(20)30098-3/pdf
coupled sodium reabsorption through SGLT2. This lowers sodium delivery to the macula densa, which is sensed as inadequate perfusion, leading to an increase in GFR via inhibition of tubuloglomerular feedback (TGF). Glomerular hyperfiltration, defined as a supraphysiological increase in GFR, contributes to the incidence
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