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https://www.ncbi.nlm.nih.gov/pubmed/8792065
Oxygen delivery and consumption during sepsis. ... This article discusses the oxygen consumption (VO2) and delivery (DO2) relationship as it pertains to animal models of sepsis and human sepsis syndrome and septic shock. Pathologic dependence of VO2 on DO2 is not present in resuscitated patients who have sepsis syndrome and septic shock.Author: Dean R. Chittock, James A. Russell
https://reference.medscape.com/medline/abstract/8792065
This article discusses the oxygen consumption (VO2) and delivery (DO2) relationship as it pertains to animal models of sepsis and human sepsis syndrome and septic shock. Pathologic dependence of VO2 on DO2 is not present in resuscitated patients who have sepsis syndrome and septic shock.
https://www.uptodate.com/contents/oxygen-delivery-and-consumption#!
The oxygen is then transported from the lungs to the peripheral tissues, where it is removed from the blood and used to fuel aerobic cellular metabolism. This process can be conceptualized as three steps: oxygenation, oxygen delivery, and oxygen consumption. In this topic review, oxygen delivery and consumption are reviewed.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6206202/
Oct 23, 2018 · Clinicians target global oxygen delivery (DO 2) as the therapeutic goal with the hope of matching the supply-demand gap of oxygen during the treatment of shock. In septic shock, both oxygen delivery and oxygen consumption are dysfunctional.Author: Chulananda Dais Goonasekera, Joseph A Carcillo, Akash Deep
https://www.frontiersin.org/articles/10.3389/fped.2018.00314/full
Oct 23, 2018 · We used the ratio of global oxygen consumption (VO 2) to global oxygen delivery (DO 2) standardized to body surface area i.e., VO 2 I/DO 2 I ratio (global oxygen extraction ratio - gO 2 ER, normal 0.28) as a crude clinical measure of global “efficacy” of oxygen consumption during resuscitation and stabilization phases of sepsis. We ...Author: Chulananda Dais Goonasekera, Joseph A Carcillo, Akash Deep
https://www.medscape.com/answers/168402-27316/why-is-oxygen-uptake-limited-in-sepsisseptic-shock
Nov 06, 2019 · Kreymann G, Grosser S, Buggisch P, Gottschall C, Matthaei S, Greten H. Oxygen consumption and resting metabolic rate in sepsis, sepsis syndrome, and septic shock. Crit Care Med . 1993 Jul. 21 (7 ...
https://www.atsjournals.org/doi/pdf/10.1513/AnnalsATS.201501-069CC
decreases in oxygen delivery cause tissue hypoxia and a fall in oxygen consumption. Based primarily on animal studies, this occurs when the ratio of oxygen consumption to oxygen delivery (V_ O 2/D O2), or the so-called oxygen extraction ratio (ERO2), exceeds about 0.7. This is referred to as the critical extraction ratio (ERO2crit).
https://www.atsjournals.org/doi/full/10.1513/AnnalsATS.201501-069CC
Jun 01, 2015 · There is, however, a critical threshold above which oxygen extraction cannot be increased, and further decreases in oxygen delivery cause tissue hypoxia and a fall in oxygen consumption. Based primarily on animal studies, this occurs when the ratio of oxygen consumption to oxygen delivery (V˙ o 2 /D O2 ), or the so-called oxygen extraction ...Author: Roberts Jk, Disselkamp M, Yataco Ac
https://link.springer.com/chapter/10.1007/978-3-642-79224-3_19
Abstract. The concept of increasing oxygen delivery (DO 2) has been applied in various diseases and syndromes, including sepsis, in order to correct impaired oxygen transport to tissues, possible mismatch between regional blood flow and regional oxygen demand, or altered tissue capability for …Author: L. Gattinoni, L. Brazzi, P. Pelosi
https://journal.chestnet.org/article/S0012-3692(15)40224-7/fulltext
The effects of increasing oxygen delivery on oxygen consumption in eight patients with septic shock and five patients with hypovolemic shock were studied during fluid resuscitation. In the septic shock group, increased from 315 ± 29 to 424 ± 25 ml/min/m2 (p <0.01) and increased from 134 ± 8 to 151 ± 7 ml/min/m2 (p <0.01). In the hypovolemic shock group, increased from 239 ± 26 to 386 ...Author: Brian S. Kaufman, Eric C. Rackow, Jay L. Falk
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